Sequence variants of α‐methylacyl‐CoA racemase are associated with prostate cancer risk: A replication study in an ethnically homogeneous population
Identifieur interne : 008745 ( Main/Exploration ); précédent : 008744; suivant : 008746Sequence variants of α‐methylacyl‐CoA racemase are associated with prostate cancer risk: A replication study in an ethnically homogeneous population
Auteurs : Liesel M. Fitzgerald [Australie] ; Russell Thomson [Australie] ; Andrea Polanowski [Australie] ; Briony Patterson [Australie] ; James D. Mckay [France] ; James Stankovich [Australie] ; Joanne L. Dickinson [Australie]Source :
- The Prostate [ 0270-4137 ] ; 2008-09-15.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Gynécologie, Génétique, Homme.
English descriptors
- KwdEn :
- Allele, Allele frequencies, Amacr, Amacr gene, Amacr gene variants, Amacr genevariants, Amacr variants, Andrology, Beckman coulter, Blood samples, Buccal mucosa swabs, Cancer council, Critical role, Dataset, Exact test, Familial, Familial cases, Familial prostate cancer, Familial prostate cancer cases, Further evidence, Gene, Genet, Genetic heterogeneity, Genetic variability, Genetics, Genotype, Genotype frequencies, Greater frequency, Gynecology, Haplotype, Haplotype analysis, Hereditary prostate cancer, Homogeneous medium, Human, Initial denaturation, Localized prostate cancer, Menzies research institute, Minor alleles, Mqls, Mqls analysis, Mqls test, Multiple cases, Nephrology, Nonclassic site, Odds ratio, Polymorphism, Population, Primer, Prostate, Prostate cancer, Prostate cancer cases, Prostate cancer dataset, Prostate cancer risk, Racemase, Racemase expression, Racemase gene, Replication, Risk factor, Sequence variants, Sequencing, Sequencing reactions, Snp, Sporadic cases, Tasmanian, Tasmanian population, Transcription factor binding sites, Unaffected controls, Variant, α-Methylacyl-CoA racemase.
- Teeft :
- Allele, Allele frequencies, Amacr, Amacr gene, Amacr gene variants, Amacr genevariants, Amacr variants, Beckman coulter, Blood samples, Buccal mucosa swabs, Cancer council, Critical role, Dataset, Exact test, Familial, Familial cases, Familial prostate cancer, Familial prostate cancer cases, Further evidence, Genet, Genetic heterogeneity, Genotype, Genotype frequencies, Greater frequency, Haplotype, Haplotype analysis, Hereditary prostate cancer, Initial denaturation, Localized prostate cancer, Menzies research institute, Minor alleles, Mqls, Mqls analysis, Mqls test, Multiple cases, Nonclassic site, Odds ratio, Polymorphism, Primer, Prostate, Prostate cancer, Prostate cancer cases, Prostate cancer dataset, Prostate cancer risk, Racemase, Racemase expression, Racemase gene, Sequence variants, Sequencing, Sequencing reactions, Snp, Sporadic cases, Tasmanian, Tasmanian population, Transcription factor binding sites, Unaffected controls, Variant.
Abstract
Examination of variants of the α‐methylacyl‐CoA racemase (AMACR) gene, as genetic contributors to prostate cancer risk, has been of considerable interest given the gene's recently established role as a diagnostic biomarker for prostate cancer.
Url:
DOI: 10.1002/pros.20798
Affiliations:
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unit="page-count">7</biblScope><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2008-09-15">2008-09-15</date></imprint><idno type="ISSN">0270-4137</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0270-4137</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Allele</term><term>Allele frequencies</term><term>Amacr</term><term>Amacr gene</term><term>Amacr gene variants</term><term>Amacr genevariants</term><term>Amacr variants</term><term>Andrology</term><term>Beckman coulter</term><term>Blood samples</term><term>Buccal mucosa swabs</term><term>Cancer council</term><term>Critical role</term><term>Dataset</term><term>Exact test</term><term>Familial</term><term>Familial cases</term><term>Familial prostate cancer</term><term>Familial prostate cancer cases</term><term>Further evidence</term><term>Gene</term><term>Genet</term><term>Genetic heterogeneity</term><term>Genetic variability</term><term>Genetics</term><term>Genotype</term><term>Genotype frequencies</term><term>Greater frequency</term><term>Gynecology</term><term>Haplotype</term><term>Haplotype analysis</term><term>Hereditary prostate cancer</term><term>Homogeneous medium</term><term>Human</term><term>Initial denaturation</term><term>Localized prostate cancer</term><term>Menzies research institute</term><term>Minor alleles</term><term>Mqls</term><term>Mqls analysis</term><term>Mqls test</term><term>Multiple cases</term><term>Nephrology</term><term>Nonclassic site</term><term>Odds ratio</term><term>Polymorphism</term><term>Population</term><term>Primer</term><term>Prostate</term><term>Prostate cancer</term><term>Prostate cancer cases</term><term>Prostate cancer dataset</term><term>Prostate cancer risk</term><term>Racemase</term><term>Racemase expression</term><term>Racemase gene</term><term>Replication</term><term>Risk factor</term><term>Sequence variants</term><term>Sequencing</term><term>Sequencing reactions</term><term>Snp</term><term>Sporadic cases</term><term>Tasmanian</term><term>Tasmanian population</term><term>Transcription factor binding sites</term><term>Unaffected controls</term><term>Variant</term><term>α-Methylacyl-CoA racemase</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Andrologie</term><term>Cancer de la prostate</term><term>Facteur risque</term><term>Gynécologie</term><term>Gène</term><term>Génotype</term><term>Génétique</term><term>Homme</term><term>Milieu homogène</term><term>Néphrologie</term><term>Population</term><term>Réplication</term><term>Variabilité génétique</term><term>Variant</term><term>α-Methylacyl-CoA racemase</term></keywords><keywords scheme="Teeft" xml:lang="en"><term>Allele</term><term>Allele frequencies</term><term>Amacr</term><term>Amacr gene</term><term>Amacr gene variants</term><term>Amacr genevariants</term><term>Amacr variants</term><term>Beckman coulter</term><term>Blood samples</term><term>Buccal mucosa swabs</term><term>Cancer council</term><term>Critical role</term><term>Dataset</term><term>Exact test</term><term>Familial</term><term>Familial cases</term><term>Familial prostate cancer</term><term>Familial prostate cancer cases</term><term>Further evidence</term><term>Genet</term><term>Genetic heterogeneity</term><term>Genotype</term><term>Genotype frequencies</term><term>Greater frequency</term><term>Haplotype</term><term>Haplotype analysis</term><term>Hereditary prostate cancer</term><term>Initial denaturation</term><term>Localized prostate cancer</term><term>Menzies research institute</term><term>Minor alleles</term><term>Mqls</term><term>Mqls analysis</term><term>Mqls test</term><term>Multiple cases</term><term>Nonclassic site</term><term>Odds ratio</term><term>Polymorphism</term><term>Primer</term><term>Prostate</term><term>Prostate cancer</term><term>Prostate cancer cases</term><term>Prostate cancer dataset</term><term>Prostate cancer risk</term><term>Racemase</term><term>Racemase expression</term><term>Racemase gene</term><term>Sequence variants</term><term>Sequencing</term><term>Sequencing reactions</term><term>Snp</term><term>Sporadic cases</term><term>Tasmanian</term><term>Tasmanian population</term><term>Transcription factor binding sites</term><term>Unaffected controls</term><term>Variant</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Gynécologie</term><term>Génétique</term><term>Homme</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract">Examination of variants of the α‐methylacyl‐CoA racemase (AMACR) gene, as genetic contributors to prostate cancer risk, has been of considerable interest given the gene's recently established role as a diagnostic biomarker for prostate cancer.</div></front></TEI><affiliations><list><country><li>Australie</li><li>France</li></country><region><li>Auvergne-Rhône-Alpes</li><li>Rhône-Alpes</li><li>Victoria (État)</li></region><settlement><li>Lyon</li><li>Melbourne</li></settlement></list><tree><country name="Australie"><noRegion><name sortKey="Fitzgerald, Liesel M" sort="Fitzgerald, Liesel M" uniqKey="Fitzgerald L" first="Liesel M." last="Fitzgerald">Liesel M. Fitzgerald</name></noRegion><name sortKey="Dickinson, Joanne L" sort="Dickinson, Joanne L" uniqKey="Dickinson J" first="Joanne L." last="Dickinson">Joanne L. Dickinson</name><name sortKey="Dickinson, Joanne L" sort="Dickinson, Joanne L" uniqKey="Dickinson J" first="Joanne L." last="Dickinson">Joanne L. Dickinson</name><name sortKey="Dickinson, Joanne L" sort="Dickinson, Joanne L" uniqKey="Dickinson J" first="Joanne L." last="Dickinson">Joanne L. Dickinson</name><name sortKey="Patterson, Briony" sort="Patterson, Briony" uniqKey="Patterson B" first="Briony" last="Patterson">Briony Patterson</name><name sortKey="Polanowski, Andrea" sort="Polanowski, Andrea" uniqKey="Polanowski A" first="Andrea" last="Polanowski">Andrea Polanowski</name><name sortKey="Stankovich, James" sort="Stankovich, James" uniqKey="Stankovich J" first="James" last="Stankovich">James Stankovich</name><name sortKey="Stankovich, James" sort="Stankovich, James" uniqKey="Stankovich J" first="James" last="Stankovich">James Stankovich</name><name sortKey="Thomson, Russell" sort="Thomson, Russell" uniqKey="Thomson R" first="Russell" last="Thomson">Russell Thomson</name></country><country name="France"><region name="Auvergne-Rhône-Alpes"><name sortKey="Mckay, James D" sort="Mckay, James D" uniqKey="Mckay J" first="James D." last="Mckay">James D. Mckay</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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